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Funded Studies

The Foundation supports research across basic, translational and clinical science to speed breakthroughs that can lead to the creation of new treatments and a better quality of life for people with Parkinson's disease.

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Previously funded studies appear chronologically, with the most recent appearing first.

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  • Alpha-synuclein Pathology, 2014
    Characterization and Validation of Disease-specific Alpha-synuclein Species in Cerebrospinal Fluid

    Study Rationale:                   
    The protein alpha-synuclein plays a major role in Parkinson’s disease (PD). Quantification of alpha-synuclein in biological fluids (e.g., cerebrospinal fluid) could...

  • LRRK2 Challenge, 2013
    LRRK2 and Vitamin D3 Metabolism

    Objective/Rationale:             
    The LRRK2 kinase enzyme is mutated in some patients with Parkinson’s disease (PD). Coincidentally, the kinase is widely expressed in most organs of the body. We...

  • Clinical Intervention Awards, 2013
    Dipraglurant for Levodopa-Induced Dyskinesia Pre-phase IIB Studies

    Study Rationale:                   
    Dipraglurant is a modulator of the metabotropic glutamate receptor 5 (mGluR5). Blockade of this receptor has been shown to have anti-parkinsonian and anti-dyskinetic...

  • MJFF Research Grant, 2012
    Novel PD Tools: Generation of Posttranslationally Modified Alpha-synuclein

    Objective/Rationale:             
    There is increasing evidence linking specific posttranslational modification (folding, cutting, etc. of proteins before maturation) and alpha-synuclein aggregation...

  • MJFF Research Grant, 2011
    Generation of Synthetic Alpha-synuclein for the PD Research Community

    Objective/Rationale:
    Despite increasing evidence linking specific modifications to alpha-synuclein (alpha-syn), alpha-synuclein aggregation, and Lewy body (LB) formation, little is still known...

  • LRRK2 Challenge, 2011
    Regulation of LRRK2 Activity by Nitric Oxide-Mediated Protein S-Nitrosylation

    Objective/Rationale:
    Mutations in the LRRK2 gene are a common cause of familial Parkinson’s disease (PD). Familial mutations can influence the GTPase or kinase activity of LRRK2 which may be...

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