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Funded Studies

The Foundation supports research across basic, translational and clinical science to speed breakthroughs that can lead to the creation of new treatments and a better quality of life for people with Parkinson's disease.

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Previously funded studies appear chronologically, with the most recent appearing first.

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  • Community Fast Track, 2003
    The regulation of synaptic dopamine by striatal nicotinic receptors

    A new category of drugs that offer a promising therapy for Parkinson's disease are drugs that act at a key subtype of brain receptors for the neurotransmitter acetylcholine, the nicotine-sensitive...

  • Community Fast Track, 2003
    Intracellular Trafficking Dysfunction Caused by Alpha-synuclein Aggregation

    A growing body of evidence suggests that aggregation of a neuronal protein named a-synuclein might be the fundamental cause of many neurodegenerative diseases, such as Parkinson's disease (PD)...

  • Community Fast Track, 2003
    Analysis of genetic factors influencing alpha-synuclein toxicity

    Parkinson's disease is a devastating disease that robs its victims of the ability to direct and control normal movement. These symptoms result from the death of a particular class of nerve cell in the...

  • Community Fast Track, 2003
    Adenosine A2A Receptors in a Pre-clinical Model of PD Dyskinesias

    Gradual slowing of movement in Parkinson's disease (PD) is due to the steady decline in brain levels of dopamine, a chemical signal that facilitates normal movement. Our most effective medications for...

  • Community Fast Track, 2003
    Morphology Specific Antibodies as Potential Therapeutics

    Parkinson's Disease has been correlated with the over expression and misfolding of the protein a-synuclein. This protein is abundantly produced in nerve cells in the brain and is the major component...

  • Community Fast Track, 2003
    Anti-inflammatory Effects of Nicotine in Parkinson’s Disease

    Parkinson's disease (PD) is a progressive, debilitating movement disorder resulting from the death of the dopamine-synthesizing neurons and, consequently, dopamine deficiency in the basal ganglia, i.e...

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