Benjamin Dehay received his PhD from the University of Paris VI in 2007, where he studied Huntington’s disease. He then undertook postdoctoral training in the laboratory of Dr. Miquel Vila, where his work mainly focused on the molecular mechanisms of neuronal cell death and the mechanism and significance of Lewy body formation in Parkinson’s disease (PD). Since 2010, Dr. Dehay has worked at the Institute of Neurodegenerative Diseases Research Group of Dr. Erwan Bezard, first as a postdoctoral fellow and later (2013) as a principal investigator. The main focus of Dr. Dehay’s research is studying the role of lysosomal dysfunction in PD and developing new neuroprotective and disease-modifying therapeutic strategies to restore lysosomal levels and function. His group also uses preclinical models to explore the progressive spreading of alpha-synuclein aggregates.
Associated Grants
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Investigating the Role of Lysosomal Biomarker ATP13A2 in the Pathogenesis of Parkinson’s Disease
2023
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SUPPLEMENT | CMA as a Means to Counteract Alpha-synuclein Pathology in Non-human Primates”
2023
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