The Foundation supports research across basic, translational and clinical science to speed breakthroughs that can lead to the creation of new treatments and a better quality of life for people with Parkinson's disease.
Search or browse funded studies
Previously funded studies appear chronologically, with the most recent appearing first.
-
Community Fast Track, 2004Vaccine-induced Neuroprotection in an Animal Model of Parkinson's Disease
There is a growing appreciation that immune responses can significantly improve neuronal survival after injury or in neurodegenerative diseases. Immune-deficient mice have reduced neurorepair...
-
The Role of Inflammation in Parkinson's Disease, 2003Inflammatory Mechanisms & PD Risk: A Discordant Twin Pairs Case-Control Study
The cause of Parkinson’s disease is unknown. A genetic cause is identified in only a small percentage of cases. We believe that environmental factors play an important role in most cases of PD. While...
-
Molecular Mechanisms of Dyskinesia in Parkinson's Disease, 2003The Role of Abnormal Gap Junction Communication in the Generation of Levodopa-induced Dyskinesia in Parkinson's Disease
We hypothesize that enhanced gap junction communication (GJC) plays an unheralded and important role in the neural mechanisms underlying levodopa-induced dyskinesia (LID). Gap junctions are a special...
-
Molecular Mechanisms of Dyskinesia in Parkinson's Disease, 2003A Trial of Low Frequency Repetitive Transcranial Magnetic Stimulation for Treatment of Dyskinesia
Levodopa-induced dyskinesia (LID) is a major source of disability of many patients with Parkinson's disease (PD). LID may be associated with increased activity of the part of the brain that mediates...
-
Molecular Mechanisms of Dyskinesia in Parkinson's Disease, 2003Anatomical and neurochemical characterization of basal ganglia neuropeptides in levodopainduceddyskinesia
The most common treatment for Parkinson's disease is levodopa (L-DOPA). This usually gives good relief of symptom for many years but, eventually, complications such as abnormal involuntary movements...
-
Molecular Mechanisms of Dyskinesia in Parkinson's Disease, 2003Reversal of Dyskinesias By in-vivo Gene Transfer of L-DOPA Using Adeno-associated Viral Vectors
Parkinson's disease is a result of an extensive loss of the pigmented neurons located in the brain stem (termed the Substantia Nigra). These nerve cells are the principal neurons producing dopamine in...
Apply for a Grant
Our funding programs support basic, translational and clinical research from academia and industry.